CANCER BIOLOGY & THERAPY - Journal - Bridge of Knowledge

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CANCER BIOLOGY & THERAPY

ISSN:

1538-4047

eISSN:

1555-8576

Disciplines
(Field of Science):

  • biomedical engineering (Engineering and Technology)
  • medical biology (Medical and Health Sciences )
  • pharmacology and pharmacy (Medical and Health Sciences )
  • medical sciences (Medical and Health Sciences )
  • health sciences (Medical and Health Sciences )
  • biotechnology (Natural sciences)
  • biological sciences (Natural sciences)
  • chemical sciences (Natural sciences)

Ministry points: Help

Ministry points - current year
Year Points List
Year 2024 100 Ministry scored journals list 2024
Ministry points - previous years
Year Points List
2024 100 Ministry scored journals list 2024
2023 100 Ministry Scored Journals List
2022 100 Ministry Scored Journals List 2019-2022
2021 100 Ministry Scored Journals List 2019-2022
2020 100 Ministry Scored Journals List 2019-2022
2019 100 Ministry Scored Journals List 2019-2022
2018 30 A
2017 30 A
2016 30 A
2015 30 A
2014 30 A
2013 30 A
2012 30 A
2011 30 A
2010 27 A

Model:

Open Access

Points CiteScore:

Points CiteScore - current year
Year Points
Year 2022 9.8
Points CiteScore - previous years
Year Points
2022 9.8
2021 8.3
2020 6.2
2019 5.4
2018 6
2017 6.2
2016 5.4
2015 5.1
2014 6.5
2013 6.8
2012 5.6
2011 4.8

Impact Factor:

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total: 3

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Catalog Journals

Year 2015
  • Human UDP-Glucuronosyltransferases: Effects of altered expression in breast and pancreatic cancer cell lines.
    Publication
    • C. Dates
    • T. Fahmi
    • S. Pyrek
    • A. Yao-Borengasser
    • B. Borowa-Mazgaj
    • S. M. Bratton
    • S. Kadlubar
    • P. Mackenzie
    • R. Haun
    • A. Radominska-Pandya

    - CANCER BIOLOGY & THERAPY - Year 2015

    Increased aerobic glycolysis and de novo lipid biosynthesis are common characteristics of invasive cancers. UDP-glucuronosyltransferases (UGTs) are phase II drug metabolizing enzymes that in normal cells possess the ability to glucuronidate these lipids and speed their excretion; however, de-regulation of these enzymes in cancer cells can lead to an accumulation of bioactive lipids, which further fuels cancer progression. We hypothesize...

    Full text available to download

Year 2011
Year 2008

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